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Growth hormone regulation of p85alpha expression and phosphoinositide 3-kinase activity in adipose tissue: mechanism for growth hormone-mediated insulin resistance

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It may promote abnormal angiogenesis (new blood-vessel growth), raising theoretical concerns in patients with cancer history

Acknowledgements We thank all the researchers and clinicians whose work contributed to the body of evidence reviewed in this manuscript

in the other outcome, the injured tissue continues to deteriorate, and the mechanism of the persisting damage involves gliosis with the loss of support of neurotrophic factors, persistent inflammatory receptor activation, and changes in microglia and astrocytes that continue to release harmful cytokines, which promote neuronal death and axonal injury

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