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has reported that administration of IKK inhibitors BAY-65-1942 inhibited the microglial activation, attenuated the loss of dopamine neurons, and enhanced levels of neurotransmitters [71], and the similar result was observed after administration of IKK inhibitors, such as BAY-65-1942, metformin, and ginsenoside Rb1, and peptide [70, 74, 75]

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Unlike the gastric phase, the intestinal phase showed an increased PDI for EMNPs, nearing 0.50, suggesting a polydisperse distribution and nanoparticle aggregation (Supplementary Fig

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