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doi: 10.1038/gim.2016.202 8

Specifically, the hypoxic synovial milieu initially induces lipid synthesis in T cells, and this metabolic reprogramming in turn drives pathogenic T cell phenotypes and enhances tissue infiltration capacity

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This complexity makes identifying and specifically targeting the most relevant pathways within this vast network difficult, as targeting a single oxidant or source may be insufficient and could potentially interfere with physiological redox signaling, given ROSs crucial dual roles as signaling molecules (as elaborated in Section 2.2) (Cheng et al., 2024)

Most of these studies have been on animals, so there is more research needed on humans