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Importantly, we provide novel evidence that prolonged systemic treatment with NAC induces cell subtype-specific changes in synaptic transmission in the NuAc core: quantal glutamatergic currents were larger in D1-MSNs, while the excitatory drive onto D2-MSNs was overall diminished

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Receptor binding can either mediate the opening of ion channels or cause metabolic changes within the cell (3, 14)

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