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In parallel, a failure in the antioxidant mechanism would occur, disrupting cell homeostasis and culminating in cell damage.52 Melanocytes cultured from unaffected areas in patients with vitiligo have a greater susceptibility to oxidative stress than from controls without vitiligo, demonstrating an overall susceptibility of the patient with vitiligo to oxidative damage.53 There is an increase in the superoxide dismutase enzyme (responsible for degrading the O2 radical into H2O2 and O2), an increase in lipid peroxidation (secondary to OS), in addition to a reduction in the catalase enzyme (which converts H2O2 into H2O and O2) in vitiligo

Durner and Klessig [35] examined the effects of SA on the formation of the various redox states or reaction intermediates of catalase (Figure 3)

Pediatric research

CMC and U 13C315N-CMC, m/z 271 and 275, respectively

10.3389/fphar.2021.752148 92 QiuL.CaiZ.ZhangN

A 104 , 1127411279