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Hence, it is now urgent to discover new, effective and safe strategies to prevent and/or treat this hardly bearable condition

ILC2s are capable of both initiating and propagating many of the pathological features of allergic airways disease, potentially playing a critical role in acute disease exacerbations ( -/- mice, cultured for 24h in the presence or absence of melatonin, and then adoptively transferred the ILC2s into Rag2 -/- GC -/- mice utilizing established protocol as described by our group previously ( Figure 4A )

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