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Notably, DNA damage and subsequent genome instability are major driving forces for tumorigenesis and aging via driving mutation

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C-Src is yet another pathway that control various biological functions where its aberrant regulation can lead to anchorage-independent growth, survival, tumor vascularity, migration, metastasis, survival and invasion [93]

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This regulatory activity may support endothelial function and promote vessel relaxation, contributing to improved hemodynamic balance in experimental settings