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The majority of available experimental data on vitamin E for suppressing PCa has been obtained with -tocopherol, leaving the possibility that other vitamin E isomers may possess better chemopreventive or anticancer potential against PCa, but which needs to be explored further [28]

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Although ferroptosis and cuproptosis follow distinct regulatory mechanisms, recent studies have suggested a possible interconnection through cellular stress responses, including mitochondrial dysfunction, ROS buildup, and metabolic reprogramming

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