increased glutathione levels gliomas autophagy

increased glutathione levels gliomas autophagy and Its Metabolic Enzymes in Gliomal Tumor Tissue and the Peritumoral Zone at Different Degrees of Anaplasia Glutathione metabolism-related gene signature predicts

Name: increased glutathione levels gliomas autophagy and Its Metabolic Enzymes in Gliomal Tumor Tissue and the Peritumoral Zone at Different Degrees of Anaplasia Glutathione metabolism-related gene signature predicts
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SKU: 94183100479
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Glutathione metabolism related gene signature predicts prognosis and treatment response in low grade glioma Aging Glutamine Imaging: A New Avenue for Glioma Management American Journal of Neuroradiology Relationship between Glutathione Dependent Enzymes and the Immunohistochemical Profile of Glial Neoplasms Promising and challenging phytochemicals targeting LC3 mediated autophagy signaling in cancer therapy Rajendran 2024 Immunity, Inflammation and Disease Wiley Online Library The GlutamateGlutathione axis in neuropsychiatric disorders and cancer: From shared mechanisms to non invasive biomarkers ScienceDirect Revisiting the potential of regulated cell death in glioma treatment: a focus on autophagy dependent cell death, anoikis, ferroptosis, cuproptosis, pyroptosis, immunogenic cell death, and the crosstalk between them PMC

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The first indication that TRIMs as a family could directly intersect with the autophagy machinery was published in 2014, when TRIM5, TRIM6, TRIM17, TRIM22, and TRIM49 were shown to interact with the autophagy regulators ULK1 and Beclin 1 (Mandell et al., 2014)